American Society of Microbiology ABMM Actual Tests : ABMM

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American Society of Microbiology ABMM Exam Syllabus Topics:

American Society of Microbiology ABMM Sample Questions:

1. A research laboratory is investigating the mechanism of action of a novel antiviral drug against herpes simplex virus. The drug is found to inhibit the viral DNA polymerase. Which of the following commercially available antiviral agents has a similar mechanism of action?

A) Oseltamivir
B) Amantadine
C) Interferon-alpha
D) Acyclovir

2. A patient with a history of valvular heart disease undergoes a dental procedure and subsequently develops a bloodstream infection with Streptococcus mutans. This organism gains access to the bloodstream primarily due to:

A) Production of potent extracellular enzymes.
B) Formation of a protective biofilm on the teeth.
C) Transient bacteremia resulting from the procedure.
D) Active invasion of the oral mucosa.

3. A patient presents with a pruritic rash consisting of raised, serpiginous tracks on their skin. The patient recently returned from a Caribbean vacation and reported walking barefoot on the beach. The MOST likely causative organism is:

A) Pediculus humanus
B) Sarcoptes scabiei
C) Larvae of Ancylostoma braziliense
D) Larvae of Strongyloides stercoralis

4. A microbiology laboratory receives a blood culture from a patient with suspected bacterial endocarditis. After
24 hours of incubation, Gram-positive cocci in clusters are observed on the Gram stain. Catalase test is positive, and coagulase test is negative. Further testing reveals susceptibility to novobiocin. The MOST likely organism is:

A) Staphylococcus saprophyticus
B) Staphylococcus lugdunensis
C) Staphylococcus aureus
D) Staphylococcus epidermidis

5. A research laboratory is investigating the mechanisms of antigenic variation in Trypanosoma brucei, the causative agent of African trypanosomiasis. The parasite evades the host immune response by periodically changing its surface coat protein. This process primarily involves:

A) Segmental gene conversion events involving a large repertoire of silent VSG genes.
B) Recombination between different VSG gene loci on minichromosomes.
C) RNA interference-mediated silencing of expressed VSG genes followed by activation of a new VSG gene.
D) High-frequency point mutations in the gene encoding the variant surface glycoprotein (VSG).

Solutions: